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Health Sciences · Medicine

Neurological Complications and Syndromes
Research Guide

What is Neurological Complications and Syndromes?

Neurological Complications and Syndromes in this context refers to Posterior Reversible Encephalopathy Syndrome (PRES), a neurological condition characterized by headache, altered mental functioning, seizures, and loss of vision, with imaging findings of predominantly posterior leukoencephalopathy, often linked to eclampsia, preeclampsia, and hypertension.

PRES presents with a reversible syndrome of headache, altered mental functioning, seizures, and vision loss, associated with posterior leukoencephalopathy on imaging studies, as identified in hospitalized patients with acute illness. The field encompasses 20,143 works focused on imaging characteristics, pathophysiology, risk factors, management, and outcomes of PRES. Growth rate over the past 5 years is not available.

Topic Hierarchy

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graph TD D["Health Sciences"] F["Medicine"] S["Psychiatry and Mental health"] T["Neurological Complications and Syndromes"] D --> F F --> S S --> T style T fill:#DC5238,stroke:#c4452e,stroke-width:2px
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20.1K
Papers
N/A
5yr Growth
182.5K
Total Citations

Research Sub-Topics

Why It Matters

PRES impacts management of conditions like eclampsia, preeclampsia, and hypertension, where prompt recognition via imaging prevents permanent neurological damage. Hinchey et al. (1996) in "A Reversible Posterior Leukoencephalopathy Syndrome" described the syndrome in hospitalized patients, enabling reversal with treatment of underlying causes, with 3293 citations underscoring its clinical relevance. Scott and Smith (2009) in "Moyamoya Disease and Moyamoya Syndrome" detailed associated cerebrovascular risks, including stroke predisposition from arterial stenosis, guiding surgical interventions in affected patients.

Reading Guide

Where to Start

"A Reversible Posterior Leukoencephalopathy Syndrome" by Hinchey et al. (1996), as it provides the foundational description of clinical and imaging features in hospitalized patients, establishing PRES as a distinct entity with 3293 citations.

Key Papers Explained

Hinchey et al. (1996) in "A Reversible Posterior Leukoencephalopathy Syndrome" first characterized the core syndrome of headache, seizures, and posterior leukoencephalopathy. Scott and Smith (2009) in "Moyamoya Disease and Moyamoya Syndrome" extended understanding to associated vasculopathies predisposing to stroke, building on cerebrovascular themes with 1695 citations. Suzuki (1969) in "Cerebrovascular "Moyamoya" Disease" introduced early moyamoya concepts relevant to syndrome complications, while Suzuki and Kodama (1971) in their follow-up refined angiographic findings.

Paper Timeline

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graph LR P0["Cerebrovascular 'Moyamoya' Disease
1969 · 2.1K cites"] P1["Wegener's Granulomatosis: Prospe...
1983 · 2.0K cites"] P2["A Reversible Posterior Leukoence...
1996 · 3.3K cites"] P3["The Cytolytic P 2Z Re...
1996 · 1.7K cites"] P4["Homocysteine and Atherothrombosis
1998 · 2.0K cites"] P5["Reduced Exposure to Calcineurin ...
2007 · 1.8K cites"] P6["Moyamoya Disease and Moyamoya Sy...
2009 · 1.7K cites"] P0 --> P1 P1 --> P2 P2 --> P3 P3 --> P4 P4 --> P5 P5 --> P6 style P2 fill:#DC5238,stroke:#c4452e,stroke-width:2px
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Most-cited paper highlighted in red. Papers ordered chronologically.

Advanced Directions

Current focus remains on imaging, pathophysiology, risk factors, and outcomes of PRES, with no recent preprints or news in the last 12 months. Related cerebrovascular syndromes like moyamoya continue to inform differential diagnosis.

Papers at a Glance

# Paper Year Venue Citations Open Access
1 A Reversible Posterior Leukoencephalopathy Syndrome 1996 New England Journal of... 3.3K
2 Cerebrovascular "Moyamoya" Disease 1969 Archives of Neurology 2.1K
3 Wegener's Granulomatosis: Prospective Clinical and Therapeutic... 1983 Annals of Internal Med... 2.0K
4 Homocysteine and Atherothrombosis 1998 New England Journal of... 2.0K
5 Reduced Exposure to Calcineurin Inhibitors in Renal Transplant... 2007 New England Journal of... 1.8K
6 The Cytolytic P <sub>2Z</sub> Receptor for Extracellular ATP I... 1996 Science 1.7K
7 Moyamoya Disease and Moyamoya Syndrome 2009 New England Journal of... 1.7K
8 A Prospective Study of Plasma Homocyst(e)ine and Risk of Myoca... 1992 JAMA 1.7K
9 Cerebrovascular "Moyamoya" Disease 1971 Angiology 1.6K
10 MYCOPHENOLATE MOFETIL FOR THE PREVENTION OF ACUTE REJECTION IN... 1995 Transplantation 1.4K

Frequently Asked Questions

What is Posterior Reversible Encephalopathy Syndrome?

PRES is a reversible syndrome featuring headache, altered mental functioning, seizures, and vision loss, with imaging showing predominantly posterior leukoencephalopathy. It occurs in hospitalized patients with acute illness, often tied to eclampsia, preeclampsia, or hypertension. Early recognition allows reversal through addressing underlying triggers.

What are the clinical features of PRES?

Clinical features include headache, altered mental functioning, seizures, and loss of vision. Imaging reveals findings of predominantly posterior leukoencephalopathy. These manifestations are noted in patients hospitalized for acute illness, as reported by Hinchey et al. (1996).

How is PRES associated with eclampsia and hypertension?

PRES frequently links to eclampsia, preeclampsia, and hypertension as precipitating conditions. Vasoconstriction and imaging characteristics highlight these associations in the syndrome's pathophysiology. Management targets blood pressure control to achieve reversibility.

What role does imaging play in diagnosing PRES?

Imaging demonstrates predominantly posterior leukoencephalopathy, confirming PRES diagnosis. Characteristics include reversible changes in posterior brain regions. This aids differentiation from stroke or other neurological complications.

What are outcomes for patients with PRES?

Outcomes are often reversible with prompt treatment of underlying conditions like hypertension. Pathophysiology involves endothelial dysfunction and vasogenic edema. Long-term prognosis improves with early intervention, as evidenced in foundational studies.

Open Research Questions

  • ? What precise pathophysiological mechanisms link hypertension and eclampsia to the vasogenic edema seen in PRES?
  • ? How do imaging characteristics of PRES differ across risk factors such as immunosuppression versus preeclampsia?
  • ? Which management strategies optimize reversibility and prevent recurrent PRES episodes?
  • ? What distinguishes PRES outcomes in adults versus pediatric populations with associated conditions?
  • ? How does vasoconstriction contribute to the diverse radiological features of PRES?

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