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Inflammatory Biomarkers in Disease Prognosis
Research Guide

What is Inflammatory Biomarkers in Disease Prognosis?

Inflammatory biomarkers in disease prognosis are measurable indicators of systemic inflammation, such as neutrophil-to-lymphocyte ratio and C-reactive protein, that predict outcomes including survival and treatment response in cancers and other inflammatory conditions.

The field encompasses 45,363 papers examining inflammation's role in cancer progression, immunity, and metastasis through markers like neutrophil-to-lymphocyte ratio, platelets, and systemic immune-inflammation index. Templeton et al. (2014) in "Prognostic Role of Neutrophil-to-Lymphocyte Ratio in Solid Tumors: A Systematic Review and Meta-Analysis" found high neutrophil-to-lymphocyte ratio associated with adverse overall survival across solid tumors. Hu et al. (2014) in "Systemic Immune-Inflammation Index Predicts Prognosis of Patients after Curative Resection for Hepatocellular Carcinoma" developed the systemic immune-inflammation index from lymphocyte, neutrophil, and platelet counts to forecast hepatocellular carcinoma prognosis.

Topic Hierarchy

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graph TD D["Health Sciences"] F["Medicine"] S["Oncology"] T["Inflammatory Biomarkers in Disease Prognosis"] D --> F F --> S S --> T style T fill:#DC5238,stroke:#c4452e,stroke-width:2px
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45.4K
Papers
N/A
5yr Growth
505.6K
Total Citations

Research Sub-Topics

Why It Matters

Inflammatory biomarkers enable risk stratification and guide clinical decisions in oncology by predicting survival post-resection or chemotherapy response. Templeton et al. (2014) meta-analysis of solid tumors showed high neutrophil-to-lymphocyte ratio linked to worse overall survival, supporting its integration into prognostic scores since it is inexpensive and readily available from blood tests. Hu et al. (2014) demonstrated the systemic immune-inflammation index outperformed other markers in predicting recurrence-free survival in 133 hepatocellular carcinoma patients after curative resection, with higher index values correlating to poorer outcomes. Mantovani et al. (2008) in "Cancer-related inflammation" established inflammation as a driver of tumor progression, informing therapies targeting inflammatory pathways like those involving platelets and the tumor microenvironment.

Reading Guide

Where to Start

"Cancer-related inflammation" by Mantovani et al. (2008) provides the foundational overview of inflammation's mechanistic role in cancer, making it accessible for initial understanding before diving into specific biomarkers.

Key Papers Explained

Mantovani et al. (2008) "Cancer-related inflammation" establishes core mechanisms, which Colotta et al. (2009) "Cancer-related inflammation, the seventh hallmark of cancer: links to genetic instability" extends to genetic instability. Templeton et al. (2014) "Prognostic Role of Neutrophil-to-Lymphocyte Ratio in Solid Tumors: A Systematic Review and Meta-Analysis" quantifies neutrophil-to-lymphocyte ratio's broad applicability, while Hu et al. (2014) "Systemic Immune-Inflammation Index Predicts Prognosis of Patients after Curative Resection for Hepatocellular Carcinoma" applies a composite index to liver cancer. Diakos et al. (2014) "Cancer-related inflammation and treatment effectiveness" connects these to therapy outcomes.

Paper Timeline

100%
graph LR P0["Circulating Endothelial Progenit...
2003 · 3.4K cites"] P1["Cancer-related inflammation
2008 · 11.1K cites"] P2["Cancer-related inflammation, the...
2009 · 2.8K cites"] P3["Prognostic Role of Neutrophil-to...
2014 · 3.1K cites"] P4["The evaluation of tumor-infiltra...
2014 · 3.0K cites"] P5["Role of C-Reactive Protein at Si...
2018 · 2.8K cites"] P6["Endothelial cell infection and e...
2020 · 6.5K cites"] P0 --> P1 P1 --> P2 P2 --> P3 P3 --> P4 P4 --> P5 P5 --> P6 style P1 fill:#DC5238,stroke:#c4452e,stroke-width:2px
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Most-cited paper highlighted in red. Papers ordered chronologically.

Advanced Directions

Current research builds on neutrophil-to-lymphocyte ratio and systemic immune-inflammation index for personalized prognosis in solid tumors, with focus on integrating platelets and tumor microenvironment markers. No recent preprints available, but extensions from top papers target immunotherapy response prediction.

Papers at a Glance

# Paper Year Venue Citations Open Access
1 Cancer-related inflammation 2008 Nature 11.1K
2 Endothelial cell infection and endotheliitis in COVID-19 2020 The Lancet 6.5K
3 Circulating Endothelial Progenitor Cells, Vascular Function, a... 2003 New England Journal of... 3.4K
4 Prognostic Role of Neutrophil-to-Lymphocyte Ratio in Solid Tum... 2014 JNCI Journal of the Na... 3.1K
5 The evaluation of tumor-infiltrating lymphocytes (TILs) in bre... 2014 Annals of Oncology 3.0K
6 Cancer-related inflammation, the seventh hallmark of cancer: l... 2009 Carcinogenesis 2.8K
7 Role of C-Reactive Protein at Sites of Inflammation and Infection 2018 Frontiers in Immunology 2.8K
8 Systemic Immune-Inflammation Index Predicts Prognosis of Patie... 2014 Clinical Cancer Research 2.1K
9 Pyroptosis: mechanisms and diseases 2021 Signal Transduction an... 2.1K
10 Cancer-related inflammation and treatment effectiveness 2014 The Lancet Oncology 2.1K

Frequently Asked Questions

What is the prognostic role of neutrophil-to-lymphocyte ratio in solid tumors?

High neutrophil-to-lymphocyte ratio indicates adverse overall survival in many solid tumors, as shown in a systematic review and meta-analysis by Templeton et al. (2014). This ratio is a simple, inexpensive biomarker derived from routine blood counts. Its addition to existing prognostic models merits further clinical validation.

How does systemic immune-inflammation index predict hepatocellular carcinoma outcomes?

The systemic immune-inflammation index, calculated from lymphocyte, neutrophil, and platelet counts, predicts prognosis after curative resection for hepatocellular carcinoma. Hu et al. (2014) found it superior to other indices in a study of 133 patients. Higher values correlated with worse recurrence-free and overall survival.

What is the role of C-reactive protein in inflammation sites?

C-reactive protein increases up to 1,000-fold at infection or inflammation sites as an acute-phase reactant. Sproston and Ashworth (2018) described its dissociation into monomers at these locations. This property aids in local immune responses and disease monitoring.

How does cancer-related inflammation influence tumor progression?

Cancer-related inflammation contributes to genetic instability and acts as a hallmark linking immunity to tumor development. Mantovani et al. (2008) in "Cancer-related inflammation" outlined its mechanisms in cancer progression. Colotta et al. (2009) in "Cancer-related inflammation, the seventh hallmark of cancer: links to genetic instability" connected it to increased cancer risk in inflamed tissues.

What methods evaluate tumor-infiltrating lymphocytes in breast cancer?

Tumor-infiltrating lymphocytes are assessed in breast cancer using standardized pathological evaluation methods. Salgado et al. (2014) in "The evaluation of tumor-infiltrating lymphocytes (TILs) in breast cancer: recommendations by an International TILs Working Group 2014" provided international guidelines. Higher TIL levels associate with improved prognosis in certain subtypes.

Why are platelets involved in cancer metastasis prognosis?

Platelets contribute to systemic inflammatory responses that promote metastasis in cancer. The field highlights platelets alongside neutrophil-to-lymphocyte ratio in prognostic models. Hu et al. (2014) incorporated platelet counts into the systemic immune-inflammation index for hepatocellular carcinoma.

Open Research Questions

  • ? How can combinations of inflammatory biomarkers like neutrophil-to-lymphocyte ratio and systemic immune-inflammation index improve precision in multi-tumor prognostic models?
  • ? What molecular pathways link platelet activation in inflammation to specific metastatic sites in solid tumors?
  • ? In which cancer subtypes do tumor-infiltrating lymphocytes provide independent prognostic value beyond standard clinicopathological factors?
  • ? How does variability in C-reactive protein dissociation at inflammation sites affect its reliability as a dynamic prognostic marker?
  • ? What thresholds optimize systemic immune-inflammation index cutoffs for diverse patient populations post-cancer resection?

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