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Life Sciences · Immunology and Microbiology

Immunotherapy and Immune Responses
Research Guide

What is Immunotherapy and Immune Responses?

Immunotherapy and Immune Responses is a research field centered on the immunobiology of dendritic cells, their roles in antigen presentation, modulation of immunity, and applications in cancer immunotherapy and vaccine development.

This field encompasses 135,447 works on dendritic cell subsets, tumor antigen presentation, immune tolerance, and vaccine adjuvants to enhance T cell immunity and cross-presentation. Dendritic cells capture antigens and induce primary immune responses, as detailed in 'Dendritic cells and the control of immunity' (Banchereau and Steinman, 1998) with 14,238 citations. Key advances include PD-1/PD-L1 checkpoint blockade showing antitumor activity in breast cancer, per 'PD-1 and PD-L1 Immune Checkpoint Blockade to Treat Breast Cancer' (Hartkopf et al., 2016) with 30,893 citations.

Topic Hierarchy

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graph TD D["Life Sciences"] F["Immunology and Microbiology"] S["Immunology"] T["Immunotherapy and Immune Responses"] D --> F F --> S S --> T style T fill:#DC5238,stroke:#c4452e,stroke-width:2px
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135.4K
Papers
N/A
5yr Growth
3.8M
Total Citations

Research Sub-Topics

Why It Matters

Immunotherapy leverages immune responses to treat cancers, with ipilimumab improving survival in metastatic melanoma patients compared to gp100 vaccine alone, as shown in 'Improved Survival with Ipilimumab in Patients with Metastatic Melanoma' (Hodi et al., 2010) involving 676 patients. Anti-PD-L1 antibodies achieved objective response rates of 6-17% and disease stabilization of 12-41% at 24 weeks in advanced cancers including non-small-cell lung cancer and melanoma, reported in 'Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer' (Brahmer et al., 2012). Dendritic cells drive these responses by presenting tumor antigens to T cells, supporting therapeutic vaccines, while recent funding like $11.5 million to UofL for new cancer immunotherapies and $3 million to University of Houston for a Cancer Immunotherapy Biomarker Core advances clinical translation.

Reading Guide

Where to Start

'Dendritic cells and the control of immunity' (Banchereau and Steinman, 1998) first, as it provides the foundational overview of dendritic cell roles in antigen presentation and immunity control, essential for understanding immunotherapy basics.

Key Papers Explained

'Dendritic cells and the control of immunity' (Banchereau and Steinman, 1998) establishes dendritic cell functions, extended by 'Immunobiology of Dendritic Cells' (Banchereau et al., 2000) on their antigen-presenting capabilities. 'PD-1 and PD-L1 Immune Checkpoint Blockade to Treat Breast Cancer' (Hartkopf et al., 2016) applies these to checkpoint inhibition, while 'Improved Survival with Ipilimumab in Patients with Metastatic Melanoma' (Hodi et al., 2010) demonstrates clinical T cell enhancement. 'Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer' (Brahmer et al., 2012) builds on this with response data across cancers; 'Oncology Meets Immunology: The Cancer-Immunity Cycle' (Chen and Mellman, 2013) integrates the cycle involving dendritic cells.

Paper Timeline

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graph LR P0["Dendritic cells and the control ...
1998 · 14.2K cites"] P1["Prospective identification of tu...
2003 · 10.3K cites"] P2["Foxp3 programs the development a...
2003 · 7.6K cites"] P3["Improved Survival with Ipilimuma...
2010 · 14.9K cites"] P4["Safety and Activity of Anti–PD-L...
2012 · 7.9K cites"] P5["Transposition of native chromati...
2013 · 6.7K cites"] P6["PD-1 and PD-L1 Immune Checkpoint...
2016 · 30.9K cites"] P0 --> P1 P1 --> P2 P2 --> P3 P3 --> P4 P4 --> P5 P5 --> P6 style P6 fill:#DC5238,stroke:#c4452e,stroke-width:2px
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Most-cited paper highlighted in red. Papers ordered chronologically.

Advanced Directions

Recent preprints highlight lung cancer immunotherapy progress, checkpoint blockade combinations, and resistance mechanisms as in 'Lung cancer immunotherapy in 2025: where we stand and what comes next?'. News covers trispecific antibodies augmenting T cell and myeloid responses, Mount Sinai's antibody-producing cell studies, and funding like $11.5M to UofL for new immunotherapies.

Papers at a Glance

# Paper Year Venue Citations Open Access
1 PD-1 and PD-L1 Immune Checkpoint Blockade to Treat Breast Cancer 2016 Breast Care 30.9K
2 Improved Survival with Ipilimumab in Patients with Metastatic ... 2010 New England Journal of... 14.9K
3 Dendritic cells and the control of immunity 1998 Nature 14.2K
4 Prospective identification of tumorigenic breast cancer cells 2003 Proceedings of the Nat... 10.3K
5 Safety and Activity of Anti–PD-L1 Antibody in Patients with Ad... 2012 New England Journal of... 7.9K
6 Foxp3 programs the development and function of CD4+CD25+ regul... 2003 Nature Immunology 7.6K
7 Transposition of native chromatin for fast and sensitive epige... 2013 Nature Methods 6.7K
8 Oncology Meets Immunology: The Cancer-Immunity Cycle 2013 Immunity 6.7K
9 Interleukin-10 and the Interleukin-10 Receptor 2001 Annual Review of Immun... 6.6K
10 Immunobiology of Dendritic Cells 2000 Annual Review of Immun... 6.6K

In the News

Code & Tools

Recent Preprints

Latest Developments

Frequently Asked Questions

What role do dendritic cells play in immunity?

Dendritic cells are antigen-presenting cells that induce primary immune responses by capturing antigens and transferring information to adaptive immune cells. 'Dendritic cells and the control of immunity' (Banchereau and Steinman, 1998) demonstrates their control over immunity. 'Immunobiology of Dendritic Cells' (Banchereau et al., 2000) details their unique ability to initiate T cell responses.

How does PD-1/PD-L1 blockade work in breast cancer?

PD-1 and PD-L1 immune checkpoint blockade treats breast cancer by blocking these proteins to restore antitumor activity. 'PD-1 and PD-L1 Immune Checkpoint Blockade to Treat Breast Cancer' (Hartkopf et al., 2016) shows impressive antitumor effects and potential for long-term disease control. This approach represents a breakthrough in malignant disease treatment.

What survival benefits does ipilimumab provide in melanoma?

Ipilimumab, alone or with gp100 vaccine, improves overall survival in previously treated metastatic melanoma patients over gp100 alone. 'Improved Survival with Ipilimumab in Patients with Metastatic Melanoma' (Hodi et al., 2010) reports these outcomes in a clinical trial. Adverse events are often reversible with treatment.

What are the response rates of anti-PD-L1 in advanced cancers?

Anti-PD-L1 antibody induces durable tumor regression with 6-17% objective response rates and 12-41% disease stabilization at 24 weeks in cancers like non-small-cell lung cancer, melanoma, and renal-cell cancer. 'Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer' (Brahmer et al., 2012) confirms these results. The therapy targets advanced solid tumors effectively.

How do regulatory T cells influence immune responses?

Foxp3 programs the development and function of CD4+CD25+ regulatory T cells, which modulate immunity. 'Foxp3 programs the development and function of CD4+CD25+ regulatory T cells' (Fontenot et al., 2003) establishes this mechanism. These cells help maintain immune tolerance.

What is the cancer-immunity cycle?

The cancer-immunity cycle describes steps from antigen release to immune effector function against tumors. 'Oncology Meets Immunology: The Cancer-Immunity Cycle' (Chen and Mellman, 2013) outlines this process. It integrates innate and adaptive immunity in oncology.

Open Research Questions

  • ? How can dendritic cell subsets be optimized for enhanced tumor antigen cross-presentation in immunotherapy?
  • ? What mechanisms underlie resistance to PD-1/PD-L1 checkpoint blockade in breast cancer and melanoma?
  • ? How does the tumor microenvironment suppress dendritic cell-induced T cell immunity?
  • ? Which adjuvants most effectively boost innate immunity for therapeutic cancer vaccines?
  • ? What factors determine the balance between immune activation and tolerance in dendritic cell responses?

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